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This Concept Map, created with IHMC CmapTools, has information related to: MCAD Deficiency, FAD+ to form FADH2, preliminary β-oxidation then enters mitochondrial matrix, NADH in order to generate ATP, acetyl CoA in order to generate ATP, acetoacetate to produce more ATP, 3-hydroxyacyl CoA oxidized by 3-hydroxyacyl CoA dehydrogenase, acetyl CoA can be used as substrate for ketogenesis, blood filtered by kidneys, FAD+ to form enoyl CoA, short-chain fatty acyl CoA enters mitochondrial matrix, acetyl CoA is not available for ketogenesis, enoyl CoA hydrated to 3-hydroxyacyl CoA, fasted state leading to low insulin:glucagon ratio, UNRESPONSIVE CHILD presents with blood glucose: 10 mg/dL, NAD+ to form 3-ketoacyl CoA, hormone-sensitive lipase mobilizing free fatty acids from adipose tissue, FAD+ to form enoyl CoA, FAD+ to form enoyl CoA, gluconeogenesis leading to severe hypoglycemia, peroxisomes to undergo preliminary β-oxidation