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This Concept Map, created with IHMC CmapTools, has information related to: Ch 7 class discussion 9.24.07, LOH is rather important for TS genes, Activated oncogenes are not compatible with fetus survival, mechanisms such as gene conversion, fusogenic agent such as non-viral related agents, care takers fix DNA mistakes, hypermethylation is exploited by G0 cells, familial requires one somatic mutation(s), gate keepers control access through the R point, hypermethylation is classified as an epigenetic event, De novo methylase makes up a new methylation pattern, sporadic requires two separate somatic mutation(s), recessive tendency for tumor formation, non-viral related agents showed a recessive tendency, familial version has an inherited mutation, inherited mutation which alone does not mean tumor formation, hypermethylation typically inactivates TS genes, methylation typically is used to make a methylation pattern, Individually mutated forms of TS genes, time to accumulate, mechanisms such as mitotic recombination