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This Concept Map, created with IHMC CmapTools, has information related to: 9.20.07 Chapter 6 class notes, Receptor deactivation is necessary to prevent cancer, Sos has been found to activate Ras, SH3 domains have a cryptic site, up-regulation of protein synthesis leads to larger than normal sized cells, β-catenins are also used to attach actin cytoskeleton to cadherins, Cell membrane to the Nucleus, Protein location changes occur during Signal transduction, PIP3 via PI3K, Broken/mutated Ras initializes apoptosis inhibition, Ras is A G-Protein, sevenless is a membrane receptor, GAPs activate the GTPase site of Ras, Receptor deactivation can occur via several mechanisms, PI3K can bind PIP2, Phospholipase C can bind PIP2, Grb2 has 2 SH3 domains, Broken/mutated Ras initializes motility, β-catenins are also used to induce transcription, phosphotyrosines and its three adjacent amino acids, PIP2 can be reformed by PTEN