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This Concept Map, created with IHMC CmapTools, has information related to: G6PD deficiency, urine causing hemoglobinurea, increased heme catabolism produces increased amounts of unconjugated bilirubin, G6PD deficiency due to deletion, G6PD deficiency can be preciptated by infection, becomes conjugated by binding to albumin and then bilirubin leaves blood and sent to liver, G6PD deficiency is clinically diagnosed by neonatal jaundice, folic acid supplement necessary in RBC synthesis, hemolytic anemia is a deficiency of RBCs, REMOVED by macrophages in spleen, favism which is hemolytic anemia induced by consumption of fava bean, deletion located on q28 locus, rigid/ less deformable RBCs so they are REMOVED by macrophages, REMOVED by macrophages in Liver, oxidant drugs such as antipyretics, bilirubin is excreted in bile, Extravascular Hemolysis predominantly seen in chronic Hemolytic episodes, iron supplements to replenish loss of iron, glucose-6-phosphate dehydrogenated to 6-phosphogluconolactone, intravascular hemolysis of RBCs causing loss of iron, insulin which also increases activity of muscle glycogen synthase