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This Concept Map, created with IHMC CmapTools, has information related to: GROUP 3 - ETIOLOGY AND PATHOGENESIS backup jic, PRISTINE GINGIVA Histologically, there is... Junctional epithelium firmly attached to the tooth surfaces, EARLY LESION Can Worsen to Established lesion if: Oral hygiene is neglected, EARLY LESION Histologically, there is 15% CT volume comprised of Inflammatory infiltrate, PRISTINE GINGIVA Host response present Little to no inflammatory infiltrate, gram negative obligate anaerobic bacteria predominates which includes Tannerella forsythia, Proinflammatory cytokines will trigger capillary action, PRISTINE GINGIVA Histologically, there is... No gingival sulcus, EARLY LESION Histologically, there is Epithelial rete pegs grow deeper to the coronal position of lesion, EARLY LESION Cells that are present during immune response are: Endothelial attachment molecule (ELAM-1), PRISTINE GINGIVA is achieved only in germ-free conditions, EARLY LESION Cells that are present during immune response are: Proinflammatory cytokines, capillary action releasing ICAM-1, gram negative obligate anaerobic bacteria predominates which includes Treponema denticola, 3 Mechanisms it inhibits the synthesis of interleukin-8 AKA local chemokine paralysis., Clinically Healthy Gingiva Histologically, there is... presence of gingival sulcus, EARLY LESION is characterized by apical movement of junctional epithelium, irreversible infectious disease characterized by predominance of gram negative microorganisms in subgingival film. Cells that are present during immune response are CD4 and CD8 that have a 1:1 ratio, irreversible infectious disease characterized by predominance of gram negative microorganisms in subgingival film. There is a microbial challenge where gram negative obligate anaerobic bacteria predominates, Clinically Healthy Gingiva Histologically, there is... increase in vascular structures, irreversible infectious disease characterized by predominance of gram negative microorganisms in subgingival film. Cells that are present during immune response are IgG4-positive B cells in the gingival tissues increase relative to IgG2-positive cells with increasing inflammation.